by Cardiff UniversityCredit: CC0 Public DomainAn expert in the communication challenges caused by dementia is using her research to inform training practices for carers and families.Professor Ali
by Cardiff UniversityCredit: CC0 Public DomainAn expert in the communication challenges caused by dementia is using her research to inform training practices for carers and families.Professor Ali
by Western Sydney UniversityCredit: Pixabay/CC0 Public DomainWestern Sydney University's NICM Health Research Institute has led a world-first clinical trial in Australia that offers new hope
by Western Sydney UniversityCredit: Pixabay/CC0 Public DomainWestern Sydney University's NICM Health Research Institute has led a world-first clinical trial in Australia that offers new hope
by Elena Garrido,Miguel Hernandez University of ElcheDiversity of Radial Glia cells in the neonatal ferret cerebral cortex, labeled with green fluorescent protein and seen within the transparent
by Elena Garrido,Miguel Hernandez University of ElcheDiversity of Radial Glia cells in the neonatal ferret cerebral cortex, labeled with green fluorescent protein and seen within the transparent
by Catherine Talbot,The ConversationCredit: AI-generated imageThe approximately850,000people living with dementia in the UK have been badly affected by COVID-19. Many live in care ho
by Catherine Talbot,The ConversationCredit: AI-generated imageThe approximately850,000people living with dementia in the UK have been badly affected by COVID-19. Many live in care ho
by American Academy of NeurologyCredit: CC0 Public DomainStiff person syndrome spectrum disorders are rare autoimmune and neurological disorders that most often cause muscle stiffness and spasms
by American Academy of NeurologyCredit: CC0 Public DomainStiff person syndrome spectrum disorders are rare autoimmune and neurological disorders that most often cause muscle stiffness and spasms
by Vanessa McMains,Johns Hopkins University(Top) The nuclear pore protein Pom121 in healthy brain cells. (Bottom) Brain cells with an ALS/dementia genetic mutation have less of the nuclear pore
by Vanessa McMains,Johns Hopkins University(Top) The nuclear pore protein Pom121 in healthy brain cells. (Bottom) Brain cells with an ALS/dementia genetic mutation have less of the nuclear pore
byUniversity of SurreyGraphical Abstract. Credit:SLEEP(2024). DOI: 10.1093/sleep/zsae193Brain waves can be manipulated while in rapid eye movement (REM) sleep, a sleep stage associat
byUniversity of SurreyGraphical Abstract. Credit:SLEEP(2024). DOI: 10.1093/sleep/zsae193Brain waves can be manipulated while in rapid eye movement (REM) sleep, a sleep stage associat
by Queen Mary, University of LondonFigure 1. Cerebral biopsy sample and fluorescent immunostaining. (A) An image of the cerebral biopsy obtained from a severe TBI patient and fixed in 10% formali
by Queen Mary, University of LondonFigure 1. Cerebral biopsy sample and fluorescent immunostaining. (A) An image of the cerebral biopsy obtained from a severe TBI patient and fixed in 10% formali
by GYMRThe future success of Alzheimer's prevention research could depend on the ability of researchers from different clinical trials to build collaborative relationships that facilitate the shar
by GYMRThe future success of Alzheimer's prevention research could depend on the ability of researchers from different clinical trials to build collaborative relationships that facilitate the shar
by Lund UniversityCredit: Unsplash/CC0 Public DomainAlzheimer's disease is characterized by the abnormal accumulation and spread of the tau protein in the brain. An international study can no
by Lund UniversityCredit: Unsplash/CC0 Public DomainAlzheimer's disease is characterized by the abnormal accumulation and spread of the tau protein in the brain. An international study can no
by Diana Yates,University of Illinois at Urbana-ChampaignWhen tested in a mouse model of Alzheimer's disease, a new compound, L1, reduced neuroinflammation, amyloid plaques and other molecul
by Diana Yates,University of Illinois at Urbana-ChampaignWhen tested in a mouse model of Alzheimer's disease, a new compound, L1, reduced neuroinflammation, amyloid plaques and other molecul